Metabolic alterations associated with T2D are very well characterised by epidemiological and research centered scientific tests. The pathogenesis and progression of T2D is ascribed to four mechanisms; greater Superior glycation conclusion merchandise (AGE) formation, elevated polyol pathway flux, activation of protein kinase C (PKC) isoforms, and improved hexosamine pathway flux[7]. https://www.directivepublications.org/journal-of-diabetology-research/